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文章名     作者     期刊名     摘要     卷     期         如果没有找到您所需要的文献，请点击 ——此处申请     共29条记录   BMP/Smad signaling is not enhanced in Hfe-deficient mice despite increased Bmp6 expression [作者：Kautz, L; Meynard, D; Besson-Fournier, C; Darnaud, V; Al Saati, T; Coppin, H; Roth, MP,期刊：Blood, 页码：2515-2520 , 文章类型： Article，,卷期：2009年114-12] Impaired regulation of hepcidin expression in response to iron loading appears to be the pathogenic mechanism for hereditary hemochromatosis. Iron normally induces expression of the BMP6 ligand, which, in turn, activates... Activation of endothelial intrinsic NF-kappa B pathway impairs protein C anticoagulation mechanism and promotes coagulation in endotoxemic mice [作者：Song, DM; Ye, XB; Xu, HL; Liu, SF,期刊：Blood, 页码：2521-2529 , 文章类型： Article，,卷期：2009年114-12] Although the role of systemic activation of the nuclear factor kappa B (NF-kappa B) pathway in septic coagulation has been well documented, little is known about the contribution of endothelial-specific NF-kappa B signal... Plerixafor (AMD3100) and granulocyte colony-stimulating factor (G-CSF) mobilize different CD34(+) cell populations based on global gene and microRNA expression signatures [作者：Donahue, RE; Jin, P; Bonifacino, AC; Metzger, ME; Ren, JQ; Wang, E; Stroncek, DF,期刊：Blood, 页码：2530-2541 , 文章类型： Article，,卷期：2009年114-12] Plerixafor (AMD3100) and granulocyte colony-stimulating factor (G-CSF) mobilize peripheral blood stem cells by different mechanisms. A rhesus macaque model was used to compare plerixafor and G-CSF-mobilized CD34(+) cells... Successful treatment of the murine model of cystinosis using bone marrow cell transplantation [作者：Syres, K; Harrison, F; Tadlock, M; Jester, JV; Simpson, J; Roy, S; Salomon, DR; Cherqui, S,期刊：Blood, 页码：2542-2552 , 文章类型： Article，,卷期：2009年114-12] Cystinosis is an autosomal recessive metabolic disease that belongs to the family of lysosomal storage disorders. The defective gene is CTNS encoding the lysosomal cystine transporter, cystinosin. Cystine accumulates in ... Nicked beta 2-glycoprotein I binds angiostatin 4.5 (plasminogen kringle 1-5) and attenuates its antiangiogenic property [作者：Nakagawa, H; Yasuda, S; Matsuura, E; Kobayashi, K; Ieko, M; Kataoka, H; Horita, T; Atsumi, T; Koike, T,期刊：Blood, 页码：2553-2559 , 文章类型： Article，,卷期：2009年114-12] Angiostatin was first discovered as a plasminogen fragment with antitumor/antiangiogenic property. One of the angiostatin isoforms, that is, angiostatin 4.5 (AS4.5), consisting of plasminogen kringle 1 to 4 and a most pa... The lymphatic tissue microenvironments in chronic lymphocytic leukemia: in vitro models and the significance of CD40-CD154 interactions [作者：Burger, JA; Gandhi, V,期刊：Blood, 页码：2560-2561 , 文章类型： Letter，,卷期：2009年114-12] Microenvironment-dependent resistance to ABT-737 in chronic lymphocytic leukemia Response [作者：Butterworth, M; Vogler, M; Dyer, MJS; Cohen, GM,期刊：Blood, 页码：2561-2562 , 文章类型： Letter，,卷期：2009年114-12] Clinical relevance of brown snake (Pseudonaja spp) factor V escaping hemostatic regulation [作者：Isbister, GK; Scorgie, FE; Seldon, M; Lincz, LF,期刊：Blood, 页码：2563-2563 , 文章类型： Letter，,卷期：2009年114-12] Clinical relevance of brown snake (Pseudonaja spp) factor V escaping hemostatic regulation Response Response [作者：Bos, MHA; Boltz, M; Pierre, LS; Masci, PP; de Jersey, J; Lavin, MF; Camire, RM,期刊：Blood, 页码：2563-2564 , 文章类型： Letter，,卷期：2009年114-12] Reduced-intensity conditioning allogeneic stem cell transplantation in HIV patients with hematologic malignancies: yes, we can [作者：Hamadani, M; Devine, SM,期刊：Blood, 页码：2564-2566 , 文章类型： Letter，,卷期：2009年114-12] New complement regulator exposed [作者：Lesher, A; Song, WC,期刊：Blood, 页码：2363-2364 , 文章类型： Editorial Material，,卷期：2009年114-12] The plasma protein CFH is a critical complement regulator with relevance to human diseases. The function of several smaller, CFH-related proteins (CFHRs) has been a mystery. In this issue of Blood, Heinen and colleagues ... Out of this nettle, danger, we must pluck this flower, safety [作者：Steensma, DP,期刊：Blood, 页码：2364-2365 , 文章类型： Editorial Material，,卷期：2009年114-12] In this issue of Blood, Greenberg and colleagues report the final results of the ECOG E1996 study, a randomized prospective clinical trial designed to assess both efficacy and safety of recombinant erythropoietin in pati... T(11q23): brand necessary [作者：Lange, BJ,期刊：Blood, 页码：2365-2366 , 文章类型： Editorial Material，,卷期：2009年114-12] A large intergroup investigation of MLL translocation partners in pediatric AML discovers low-risk star, t(1;11), confirms high-risk chromosomes 6 and 10 partners, and declares generic t11q23 insufficient for risk strati... Coagulation and innate immune responses: can we view them separately? [作者：Delvaeye, M; Conway, EM,期刊：Blood, 页码：2367-2374 , 文章类型： Article，,卷期：2009年114-12] The horseshoe crab is often referred to as a "living fossil," representative of the oldest classes of arthropods, almost identical to species in existence more than 500 million years ago. Comparative analyses of the defe... How I treat Waldenstrom macroglobulinemia [作者：Treon, SP,期刊：Blood, 页码：2375-2385 , 文章类型： Article，,卷期：2009年114-12] Waldenstrom macroglobulinemia (WM) is a distinct B-cell disorder resulting from the accumulation, predominantly in the bone marrow, of clonally related IgM-secreting lymphoplasmacytic cells. Genetic factors play an impor... Insertion of FLT3 internal tandem duplication in the tyrosine kinase domain-1 is associated with resistance to chemotherapy and inferior outcome [作者：Kayser, S; Schlenk, RF; Londono, MC; Breitenbuecher, F; Wittke, K; Du, J; Groner, S; Spath, D; Krauter, J; Ganser, A; Dohner, H; Fischer, T; Dohner, K,期刊：Blood, 页码：2386-2392 , 文章类型： Article，,卷期：2009年114-12] To evaluate internal tandem duplication (ITD) insertion sites and length as well as their clinical impact in younger adult patients with FLT3-ITD-positive acute myeloid leukemia (AML), sequencing after DNA-based amplific...