Metabotropic Regulation of RhoA/Rho-Associated Kinase by L-type Ca2+ Channels New Mechanism for Depolarization-Evoked Mammalian Arterial Contraction

[摘要]：Background: Sustained vascular smooth muscle contraction is mediated by extracellular Ca2+ influx through L-type voltage-gated Ca2+ channels (VGCC) and RhoA/Rho-associated kinase (ROCK)-dependent Ca2+ sensitization of the contractile machinery. VGCC activ

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