- Potentiation of Helicobacter pylori CagA Protein Virulence through Homodimerization
[作者:Nagase, L; Murata-Kamiya, N; Hatakeyama, M,期刊:Journal of Biological Chemistry, 页码:33622-33631 , 文章类型: Article,,卷期:2011年286-38]
- Chronic infection with Helicobacter pylori cagA-positive strains is associated with atrophic gastritis, peptic ulceration, and gastric carcinoma. The cagA gene product, CagA, is delivered into gastric epithelial cells vi...
- Cellular Content of UDP-N-acetylhexosamines Controls Hyaluronan Synthase 2 Expression and Correlates with O-Linked N-Acetylglucosamine Modification of Transcription Factors YY1 and SP1
[作者:Jokela, TA; Makkonen, KM; Oikari, S; Karna, R; Koli, E; Hart, GW; Tammi, RH; Carlberg, C; Tammi, MI,期刊:Journal of Biological Chemistry, 页码:33632-33640 , 文章类型: Article,,卷期:2011年286-38]
- Hyaluronan, a high molecular mass polysaccharide on the vertebrate cell surface and extracellular matrix, is produced at the plasma membrane by hyaluronan synthases using UDP-GlcNAc and UDP-GlcUA as substrates. The avail...
- Structure-Function Map of the Receptor Site for beta-Scorpion Toxins in Domain II of Voltage-gated Sodium Channels
[作者:Zhang, JZ; Yarov-Yarovoy, V; Scheuer, T; Karbat, I; Cohen, L; Gordon, D; Gurevitz, M; Catterall, WA,期刊:Journal of Biological Chemistry, 页码:33641-33651 , 文章类型: Article,,卷期:2011年286-38]
- Voltage-gated sodium (Na(v)) channels are the molecular targets of beta-scorpion toxins, which shift the voltage dependence of activation to more negative membrane potentials by a voltage sensor-trapping mechanism. Molec...
|