- A functional heteromeric MIF receptor formed by CD74 and CXCR4
[作者:Schwartz, V; Lue, HQ; Kraemer, S; Korbiel, J; Krohn, R; Ohl, K; Bucala, R; Weber, C; Bernhagen, J,期刊:FEBS Letters, 页码:2749-2757 , 文章类型: Article,,卷期:2009年583-17]
- MIF is a chemokine-like inflammatory mediator that triggers leukocyte recruitment by binding to CXCR2 and CXCR4. MIF also interacts with CD74/invariant chain, a single-pass membrane-receptor. We identified complexes betw...
- MCL-1ES, a novel variant of MCL-1, associates with MCL-1L and induces mitochondrial cell death
[作者:Kim, JH; Sim, SH; Ha, HJ; Ko, JJ; Lee, K; Bae, J,期刊:FEBS Letters, 页码:2758-2764 , 文章类型: Article,,卷期:2009年583-17]
- Myeloid cell leukemia-1 (MCL-1L) is a pro-survival member of the BCL-2 family that promotes cell survival. In this study, we identify a new splicing variant of human MCL-1 that encodes MCL-1ES ( extra short). Sequence an...
- Ankrd17, an ubiquitously expressed ankyrin factor, is essential for the vascular integrity during embryogenesis
[作者:Hou, SC; Chan, LW; Chou, YC; Su, CY; Chen, X; Shih, YL; Tsai, PC; Shen, CKJ; Yan, YT,期刊:FEBS Letters, 页码:2765-2771 , 文章类型: Article,,卷期:2009年583-17]
- Ankyrin repeat domain 17 (Ankrd17) encodes an ubiquitously expressed protein with two clusters of ankyrin repeats. We have used gene targeting strategy to ablate the Ankrd17 gene in mouse. The Ankrd17-deficient mice died...
- 5-Fluorotryptophan as dual probe for ground-state heterogeneity and excited-state dynamics in apoflavodoxin
[作者:Visser, NV; Westphal, AH; Nabuurs, SM; van Hoek, A; van Mierlo, CPM; Visser, AJWG; Broos, J; van Amerongen, H,期刊:FEBS Letters, 页码:2785-2788 , 文章类型: Article,,卷期:2009年583-17]
- The apoflavodoxin protein from Azotobacter vinelandii harboring three tryptophan (Trp) residues, was biosynthetically labeled with 5-fluorotryptophan (5-FTrp). 5-FTrp has the advantage that chemical differences in its mi...
- Ribavirin enhances interferon signaling via stimulation of mTOR and p53 activities
[作者:Su, WC; Liu, WL; Cheng, CW; Chou, YB; Hung, KH; Huang, WH; Wu, CL; Li, YT; Shiau, AL; Lai, MY,期刊:FEBS Letters, 页码:2793-2798 , 文章类型: Article,,卷期:2009年583-17]
- Cellular mechanisms involving the enhancement of interferon (IFN) signaling by ribavirin remain poorly understood. Here, we identified a novel role of ribavirin in the communication between p53 and the mammalian target o...
- Activation of translation via reduction by thioredoxin-thioredoxin reductase in Saccharomyces cerevisiae
[作者:Jun, KO; Song, CH; Kim, YB; An, J; Oh, JH; Choi, SK,期刊:FEBS Letters, 页码:2804-2810 , 文章类型: Article,,卷期:2009年583-17]
- Previously we reported that in vitro translation activity in extracts of Saccharomyces cerevisiae was stimulated by dithiothreitol (DTT) and further increased by the addition of thioredoxin (TRX1) [Choi, S. K. ( 2007) Th...
- CRP regulates the expression and activity of tissue factor as well as tissue factor pathway inhibitor via NF-kappa B and ERK 1/2 MAPK pathway
[作者:Chen, YX; Wang, JF; Yao, YJ; Yuan, WL; Kong, MY; Lin, YQ; Geng, DF; Nie, RQ,期刊:FEBS Letters, 页码:2811-2818 , 文章类型: Article,,卷期:2009年583-17]
- It was found that C-reactive protein (CRP) could significantly increase the expression and activity of tissue factor (TF), but decrease that of tissue factor pathway inhibitor ( TFPI) in human umbilical vein endothelial ...
- Channel-like NH3 flux by ammonium transporter AtAMT2
[作者:Neuhauser, B; Dynowski, M; Ludewig, U,期刊:FEBS Letters, 页码:2833-2838 , 文章类型: Article,,卷期:2009年583-17]
- Prokaryotes, plants and animals control ammonium fluxes by the regulated expression of ammonium transporters (AMTs) and/or the related Rhesus (Rh) proteins. Plant AMTs were previously reported to mediate electrogenic tra...
- Structural basis for the phototoxicity of the fluorescent protein KillerRed
[作者:Carpentier, P; Violot, S; Blanchoin, L; Bourgeois, D,期刊:FEBS Letters, 页码:2839-2842 , 文章类型: Article,,卷期:2009年583-17]
- The red fluorescent protein KillerRed, engineered from the hydrozoan chromoprotein anm2CP, has been reported to induce strong cytotoxicity through the chromophore assisted light inactivation (CALI) effect. Here, we prese...
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