- Cytotoxic T Cells Are the Predominant Players Providing Cross-Protective Immunity Induced by gamma-Irradiated Influenza A Viruses
[作者:Furuya, Y; Chan, J; Regner, M; Lobigs, M; Koskinen, A; Kok, T; Manavis, J; Li, P; Mullbacher, A; Alsharifi, M,期刊:Journal of virology, 页码:4212-4221 , 文章类型: Article,,卷期:2010年84-9]
- We previously demonstrated that a single dose of nonadjuvanted intranasal gamma-irradiated influenza A virus can provide robust protection in mice against both homologous and heterosubtypic challenges, including challeng...
- Rotavirus Differentially Infects and Polyclonally Stimulates Human B Cells Depending on Their Differentiation State and Tissue of Origin
[作者:Narvaez, CF; Franco, MA; Angel, J; Morton, JM; Greenberg, HB,期刊:Journal of virology, 页码:4543-4555 , 文章类型: Article,,卷期:2010年84-9]
- We have shown previously that rotavirus (RV) can infect murine intestinal B220(+) cells in vivo (M. Fenaux, M. A. Cuadras, N. Feng, M. Jaimes, and H. B. Greenberg, J. Virol. 80: 5219-5232, 2006) and human blood B cells i...
- Mechanistic Consequences of hnRNP C Binding to Both RNA Termini of Poliovirus Negative-Strand RNA Intermediates
[作者:Ertel, KJ; Brunner, JE; Semler, BL,期刊:Journal of virology, 页码:4229-4242 , 文章类型: Article,,卷期:2010年84-9]
- The poliovirus 3' noncoding region (3' NCR) is necessary for efficient virus replication. A poliovirus mutant, PV Delta 3'NCR, with a deletion of the entire 3' NCR, yielded a virus that was capable of synthesizing viral ...
- Abl Family Tyrosine Kinases Regulate Sialylated Ganglioside Receptors for Polyomavirus
[作者:Swimm, AI; Bornmann, W; Jiang, MX; Imperiale, MJ; Lukacher, AE; Kalman, D,期刊:Journal of virology, 页码:4243-4251 , 文章类型: Article,,卷期:2010年84-9]
- Sialylated lipids serve as cellular receptors for polyomaviruses. Using pharmacological inhibitors and cell lines derived from knockout mice, we demonstrate that Abl family tyrosine kinases are required for replication o...
- Initiation of Duck Hepatitis B Virus Infection Requires Cleavage by a Furin-Like Protease
[作者:Tong, YP; Tong, SP; Zhao, XA; Wang, JG; Jun, J; Park, J; Wands, J; Li, JS,期刊:Journal of virology, 页码:4569-4578 , 文章类型: Article,,卷期:2010年84-9]
- The entry mechanism of hepatitis B virus (HBV) has not been defined, and this impedes development of antiviral therapies aimed at an early step in the viral life cycle. HBV infection has both host and tissue specificitie...
- Mutational Analysis of the Herpes Simplex Virus Type 1 UL25 DNA Packaging Protein Reveals Regions That Are Important after the Viral DNA Has Been Packaged
[作者:O'Hara, M; Rixon, FJ; Stow, ND; Murray, J; Murphy, M; Preston, VG,期刊:Journal of virology, 页码:4252-4263 , 文章类型: Article,,卷期:2010年84-9]
- The herpes simplex virus type 1 (HSV-1) UL25 gene encodes a minor capsid protein, pUL25, that is essential for packaging the full-length viral genome. Six regions which contain disordered residues have been identified in...
- Novel Inhibitors of Influenza Virus Fusion: Structure-Activity Relationship and Interaction with the Viral Hemagglutinin
[作者:Vanderlinden, E; Goktas, F; Cesur, Z; Froeyen, M; Reed, ML; Russell, CJ; Cesur, N; Naesens, L,期刊:Journal of virology, 页码:4277-4288 , 文章类型: Article,,卷期:2010年84-9]
- A new class of N-(1-thia-4-azaspiro[4.5] decan-4-yl) carboxamide inhibitors of influenza virus hemagglutinin (HA)-mediated membrane fusion that has a narrow and defined structure-activity relationship was identified. In ...
- Two N-Linked Glycosylation Sites in the V2 and C2 Regions of Human Immunodeficiency Virus Type 1 CRF01_AE Envelope Glycoprotein gp120 Regulate Viral Neutralization Susceptibility to the Human Monoclonal Antibody Specific for the CD4 Binding Domain
[作者:Utachee, P; Nakamura, S; Isarangkura-na-Ayuthaya, P; Tokunaga, K; Sawanpanyalert, P; Ikuta, K; Auwanit, W; Kameoka, M,期刊:Journal of virology, 页码:4311-4320 , 文章类型: Article,,卷期:2010年84-9]
- A recombinant human monoclonal antibody, IgG1 b12 (b12), recognizes a conformational epitope on human immunodeficiency virus type 1 (HIV-1) envelope glycoprotein (Env) gp120 that overlaps the CD4 binding domain. Although...
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