- Circadian Dependence of Infarct Size and Left Ventricular Function After ST Elevation Myocardial Infarction
[作者：Reiter, R; Swingen, C; Moore, L; Henry, TD; Traverse, JH,期刊：Circulation Research, 页码：105-110 , 文章类型： Article，,卷期：2012年110-1]
- Rationale: In rodents, infarct size after ischemia/reperfusion exhibits a circadian dependence on the time of coronary occlusion. It is not known if a similar circadian dependence of infarct size occurs in humans.Objecti...
- STAT5 Activation and Cardioprotection by Remote Ischemic Preconditioning in Humans
[作者：Heusch, G; Musiolik, J; Kottenberg, E; Peters, J; Jakob, H; Thielmann, M,期刊：Circulation Research, 页码：111-115 , 文章类型： Article，,卷期：2012年110-1]
- Rationale: The heart can be protected from infarction by brief episodes of ischemia/reperfusion of a remote organ. Remote ischemic preconditioning (RIPC) by brief arm ischemia/reperfusion has been recruited in patients u...
- Inflammation in Myocardial Diseases
[作者：Marchant, DJ; Boyd, JH; Lin, DC; Granville, DJ; Garmaroudi, FS; McManus, BM,期刊：Circulation Research, 页码：126-144 , 文章类型： Review，,卷期：2012年110-1]
- Inflammatory processes underlie a broad spectrum of conditions that injure the heart muscle and cause both structural and functional deficits. In this article, we address current knowledge regarding 4 common forms of myo...
- Autoantibodies in Heart Failure and Cardiac Dysfunction
[作者：Kaya, Z; Leib, C; Katus, HA,期刊：Circulation Research, 页码：145-158 , 文章类型： Review，,卷期：2012年110-1]
- Human heart failure is a disease with multifactorial causes, considerable morbidity, and high mortality. Several circulating autoantibodies, some of them being heart-specific, play a crucial role in the progression and i...
- Activating Transcription Factor 1 Directs Mhem Atheroprotective Macrophages Through Coordinated Iron Handling and Foam Cell Protection
[作者：Boyle, JJ; Johns, M; Kampfer, T; Nguyen, AT; Game, L; Schaer, DJ; Mason, JC; Haskard, DO,期刊：Circulation Research, 页码：20-33 , 文章类型： Article，,卷期：2012年110-1]
- Rationale: Intraplaque hemorrhage (IPH) drives atherosclerosis through the dual metabolic stresses of cholesterol-enriched erythrocyte membranes and pro-oxidant heme/iron. When clearing tissue hemorrhage, macrophages are...
- Integration of Repulsive Guidance Cues Generates Avascular Zones That Shape Mammalian Blood Vessels
[作者：Meadows, SM; Fletcher, PJ; Moran, C; Xu, K; Neufeld, G; Chauvet, S; Mann, F; Krieg, PA; Cleaver, O,期刊：Circulation Research, 页码：34-46 , 文章类型： Article，,卷期：2012年110-1]
- Rationale: Positive signals, such as vascular endothelial growth factor, direct endothelial cells (ECs) to specific locations during blood vessel formation. Less is known about repulsive signal contribution to shaping ve...
- Frizzled 4 Regulates Arterial Network Organization Through Noncanonical Wnt/Planar Cell Polarity Signaling
[作者：Descamps, B; Sewduth, R; Tojias, NF; Jaspard, B; Reynaud, A; Sohet, F; Lacolley, P; Allieres, C; Lamaziere, JMD; Moreau, C; Dufourcq, P; Couffinhal, T; Duplaa, C,期刊：Circulation Research, 页码：47-58 , 文章类型： Article，,卷期：2012年110-1]
- Rationale: A growing body of evidence supports the hypothesis that the Wnt/planar cell polarity (PCP) pathway regulates endlothelial cell proliferation and angiogenesis, but the components that mediate this regulation re...
- Adrenergic Signaling Controls RGK-Dependent Trafficking of Cardiac Voltage-Gated L-Type Ca(2+) Channels Through PKD1
[作者：Jhun, BS; O-Uchi, J; Wang, WY; Ha, CH; Zhao, JJ; Kim, JY; Wong, C; Dirksen, RT; Lopes, CMB; Jin, ZG,期刊：Circulation Research, 页码：59-70 , 文章类型： Article，,卷期：2012年110-1]
- Rationale: The Rad-Gem/Kir-related family (RGKs) consists of small GTP-binding proteins that strongly inhibit the activity of voltage-gated calcium channels. Among RGKs, Rem1 is strongly and specifically expressed in car...
- Inhibition of miR-15 Protects Against Cardiac Ischemic Injury
[作者：Hullinger, TG; Montgomery, RL; Seto, AG; Dickinson, BA; Semus, HM; Lynch, JM; Dalby, CM; Robinson, K; Stack, C; Latimer, PA; Hare, JM; Olson, EN; van Rooij, E,期刊：Circulation Research, 页码：71-81 , 文章类型： Article，,卷期：2012年110-1]
- Rationale: Myocardial infarction (MI) is a leading cause of death worldwide. Because endogenous cardiac repair mechanisms are not sufficient for meaningful tissue regeneration, MI results in loss of cardiac tissue and de...
|